Avaliação do absenteísmo por motivo de doença em funcionários técnico administrativos de uma universidade federal
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Assessment of absenteeism due to illness among technical administrative employees of a federal university
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Tahir M et al. Diosmin abrogate schemically induced hepatocarcinogenesis via alleviation of oxidative stress, hyperproliferative an dinflammatory markers in murine model. Toxicology Letters 2013;220(3):205-18.
Ahmed S et al. Diosmin Modulates the NF-kB Signal Transduction Pathwaysand Downregulation of Various Oxidative Stress Markers in Alloxan Induced Diabetic Nephropathy. Inflammation 2016; 39(5):1783-97.
Tanrikulu Y et al. The protective effect of diosmin on hepatic ischemia reperfusion injury: an experimental study. Bosn. J. Basic. Med. Sci.;2013;3(4):218-24.
Queenthy SS, John B. Diosmin exhibits anti-hyperlipidemic effects in isoproterenol induced myocardial infarcted rats. Eur J Pharmacol. 2013; 718(1-3):213-8.
Senthamizhselvan, Manivannan J, Silambarasan T, Raja B. Diosmin pretreatment improves cardiac function and suppresses oxidative stress in rat heart after ischemia/reperfusion. Eur J Pharmacol. 2014; 5(736):131-7.
Arab HH, Salama SA, Omar HA, Arafa el-SA, Maghrabi IA Diosmin Protects against Ethanol-Induced Gastric Injury in Rats: Novel Anti-Ulcer Actions. PloSOne. 2015; 10(3): 1-21.
Rehman R et al. Diosmin protects against trichloroethylene-induced renal injury in Wistar rats: plausible role of p53, Bax and caspases. Br J Nutr. 2013; 110(4):699-710.
Hasan HF, Abdel-Rafei MK, Galal SM. Diosmin attenuates radiation-induced hepatic fibrosis by boosting PPAR-γ expression and hampering miR-17-5p-activated canonical Wnt-β-catenin signaling. Biochem Cell Biol. 2017; 95(3):400-414.
Jacob KD, Noren Hooten N, Trzeciak AR, Evans MK.. Markers of oxidant stress that are clinically relevant in aging and age-related disease. Mech Ageing Dev. 2013;134(3-4):139-57.
Gasparovic AC, Jaganjac M, Mihaljevic B, Sunjic SB, Zarkovic N. Assays for the measurement of lipid peroxidation. Methods Mol Biol. 2013;965:283-96.
Barbosa KBF, Costa NMB, Alfenas RCG, De Paula SO, Minim VPR, Bressan J. Estresse oxidativo: conceito, implicações e fatores modulatórios. Rev. Nutr. 2010;23(4):629-43.
Sterba M et al. Oxidative Stress, Redox Signaling, and Autophagy: Cell Death Versus Survival Antioxid Redox Signal. 20141; 21(1): 66–85.
Tsutsui H, Kinugawa S, Matsushima S. Oxidative stress and heart failure. Am J Physiol Heart Circ Physiol. 2011; 301(6):2181-90
Vasconcelos TB, Cardoso ARNR, Josino JB, Macena RHM, Macena VPD. Radicais Livres e Antioxidantes: Proteção ou Perigo? UNOPAR Científ. Ciênc. Biológ. da Saúd. 2014;16(3):213-19.
Barreiros ALBS, David JM, David JP. Estresse oxidativo: relação entre geração de espécies reativas e defesa do organismo. Quím. Nov., 2006; 29(1):113-23.
Dung TD et al. PP2A mediates diosmin p53 activation to block HA22T cell proliferation and tumor growth in xenografted nude mice through PI3K-Akt-MDM2 signaling suppression. Food Chem Toxicol. 2012;50(5):1802-10.
Srinivasan S, Pari L. Ameliorative effect of diosmin, a citrus flavonoid against streptozotocin- nicotinamide generated oxidative stress induced diabetic rats. Chem Biol Interact. 2012;195(1):43-51.
Silambarasan T, Raja B. Diosmin, a bioflavonoid reverses alterations in blood pressure, nitric oxide, lipid peroxides and antioxidant status in DOCA-salt induced hypertensive rats. Eur J Pharmacol. 2012;679(1-3):81-9.
Bondeva T, Wolf G. Reactive oxygen species in diabetic nephropathy: friend or foe? Nephrol Dial Transplant. 2014;29(11):1998-2003.
Evans JL, Goldfine ID, Maddux BA, Grodsky GM.. Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes. Endocr Rev. 2002;23(5):599-622.
Kim MJ, Lim Y. Protective effect of short-term genistein supplementation on the early stage in diabetes-induced renal damage. Mediators Inflamm. 2013;2013:1-15.
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